Fabian Gather, Dr. rer. nat.

Copyright Dr. rer. nat. Fabian Gather, Neuroanatomy and Cell Biology, MHH

Curriculum Vitae

Academic Studies

2015-2020            Doctoral studies (Dr. rer. nat.), Institute of                                               Pharmacology, Medical Center of the Johannes                                      Gutenberg University Mainz

2009-2015            Biomedical Chemistry (Diploma), Johannes                                             Gutenberg University Mainz

 

Scientific Career

since 06/2024     Group leader Neuroepigenetics and RNA-based                                      therapies (Scientist), Institute of Neuroanatomy and                                Cell Biology, Medical School Hannover

10/2020-05/2024  Postdoctoral Researcher, Developmental                                                Neuroepigenetics Lab, Institute of Anatomy and Cell Biology, University Freiburg

09/2015-02/2020  PhD student, Institute of Pharmacology, Medical Center of the JGU Mainz

07/2015-08/2015  Research Fellow, Institute of Reconstructive Neurobiology, University of Bonn, Germany

02/2013-06/2013  Research Fellow (ERASMUS internship), Bäckvall group, Department of Organic Chemistry, University of Stockholm,                                Sweden

 

Scientific Focus

  1. Investigation of transcriptional, post-transcriptional and epigenetic regulation of gene expression during neurodevelopment and     neurodegeneration
  2. Investigation and Development of new RNA-based therapies
  3. Development of cell culture and organoid models for translational and personalized research

 

Online presence

ORCID:                                                 0000-0001-9715-1582

 

Selected Publications

ZFP982 confers mouse embryonic stem cell characteristics by regulating expression of Nanog, Zfp42, and Dppa3. Dehghanian F, Bovio PP, Gather F, Probst S, Naghsh-Nilchi A, Vogel T (2024) Biochim Biophys Acta Mol Cell Res. 1871(4):119686. https://doi.org/10.1016/j.bbamcr.2024.119686


Multimodal epigenetic changes and altered NEUROD1 chromatin binding in the mouse hippocampus underlie FOXG1 syndrome. Akol I, Izzo A, Gather F, Strack S, Heidrich S, Ó hAilín D, Villarreal A, Hacker C, Rauleac T, Bella C, Fischer A, Manke T, Vogel T (2023) Proc Natl Acad Sci U S A. 120(2):e2122467120. https://doi.org/10.1073/pnas.2122467120


A specific, non-immune system-related isoform of the human inducible nitric oxide synthase is expressed during differentiation of human stem cells into various cell types. Gather F, Ihrig-Biedert I, Kohlhas P, Krutenko T, Peitz M, Brüstle O, Pautz A, Kleinert H (2022) Cell Commun Signal. 20(1):47. https//doi.org/10.1186/s12964-022-00855-x


Paving Therapeutic Avenues for FOXG1 Syndrome: Untangling Genotypes and Phenotypes from a Molecular Perspective. Akol I, Gather F, Vogel T (2022) Int J Mol Sci. 23(2):954. https//doi.org/10.3390/ijms23020954


Regulation of human inducible nitric oxide synthase expression by an upstream open reading frame. Gather F, Schmitz K, Koch K, Vogt LM, Pautz A, Kleinert H (2019) Nitric Oxide. 88:50-60. https//doi.org/10.1016/j.niox.2019.04.008 


The KH-type splicing regulatory protein (KSRP) regulates type III interferon expression post-transcriptionally.Schmidtke L, Schrick K, Saurin S, Käfer R, Gather F, Weinmann-Menke J, Kleinert H, Pautz A (2019) Biochem J. 476(2):333-352. https//doi.org/10.1042/BCJ20180522